m-Calpain Activation/Depletion is Associated With Androgen-Induced Reduction of Protein Kinase C and Proliferation of Male Accessory Sex Organ Smooth Muscles Cells

Author:

Gebrosky Norman1,Cole Dennis1,Stetter-Neel Caroline1,Durham John1,Mawhinney Michael1

Affiliation:

1. Departments of Urology, Pharmacology/Toxicology, and Biochemistry, Robert C. Byrd Health Sciences Center, Morgantown, West Virginia.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Urology

Reference20 articles.

1. Protein kinases and the androgen-induced proliferation of accessory sex organ smooth muscle;Mariotti;Biol. Reprod.,1992

2. Differential down-regulation of epidermal protein kinase C by 12-O-tetradecanoylphorbol-13-acetate and diacylglycerol: association with epidermal hyperplasia and tumor promotion;Hansen;Cancer Res.,1990

3. Protein kinase C down-regulation and non transient activation correlates with melanocyte growth;Brooks;Cancer Res.,1991

4. Phorbol esters increased the amount of Ca sup 2+, phospholipid-dependent kinase associated with plasma membrane;Kraft;Nature (Lond.),1983

5. Calcium-activated neutral protease (calpain) system: structure, function and regulation;Croall;Physiol. Rev.,1991

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