m-Calpain Activation/Depletion is Associated With Androgen-Induced Reduction of Protein Kinase C and Proliferation of Male Accessory Sex Organ Smooth Muscles Cells
Author:
Affiliation:
1. Departments of Urology, Pharmacology/Toxicology, and Biochemistry, Robert C. Byrd Health Sciences Center, Morgantown, West Virginia.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Urology
Reference20 articles.
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2. Differential down-regulation of epidermal protein kinase C by 12-O-tetradecanoylphorbol-13-acetate and diacylglycerol: association with epidermal hyperplasia and tumor promotion;Hansen;Cancer Res.,1990
3. Protein kinase C down-regulation and non transient activation correlates with melanocyte growth;Brooks;Cancer Res.,1991
4. Phorbol esters increased the amount of Ca sup 2+, phospholipid-dependent kinase associated with plasma membrane;Kraft;Nature (Lond.),1983
5. Calcium-activated neutral protease (calpain) system: structure, function and regulation;Croall;Physiol. Rev.,1991
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1. Androgen Induced Norepinephrine Release From Postganglionic Neurons Mediates Accessory Sex Organ Smooth Muscle Proliferation;Journal of Urology;2002-04
2. Androgen Induced Norepinephrine Release From Postganglionic Neurons Mediates Accessory Sex Organ Smooth Muscle Proliferation;The Journal of Urology;2002-04
3. Testosterone- and phorbol ester-stimulated proliferation in human cultured prostatic stromal cells;Cellular Signalling;2001-10
4. Differential action of nerve growth factor and phorbol ester TPA on rat synaptosomal PKC isoenzymes;Neurochemistry International;1999-10
5. PKC-dependent signalling mechanisms in differentiated smooth muscle;Acta Physiologica Scandinavica;1998-12
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