POLYMORPHISMS OF N-ACETYLTRANSFERASE 2, GLUTATHIONE S-TRANSFERASE MU AND THETA GENES AS RISK FACTORS OF BLADDER CANCER IN RELATION TO ASTHMA AND TUBERCULOSIS

Author:

KIM WUN-JAE1,LEE HYUNG-LAE1,LEE SANG-CHEOL1,KIM YONG TAE1,KIM HEON1

Affiliation:

1. From the Department of Urology and Preventive Medicine, College of Medicine, Chungbuk National University, Cheongju, Chungbuk, Korea

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Urology

Reference22 articles.

1. Role of N-acetyltransferase phenotypes in bladder carcinogenesis: a pharmacogenetic epidemiological approach to bladder cancer;Cartwright;Lancet,1982

2. White blood cell DNA adducts, smoking, and NAT2 and GSTM1 genotypes in bladder cancer: a case-control study;Peluso;Cancer Epidemiol Biomarkers Prev,1998

3. Drug-metabolizing enzymes in ligand-modulated transcription;Nebert;Biochem Pharmacol,1994

4. Genetic risk and carcinogen exposure: a common inherited defect of the cacinogen-metabolism gene glutathione S-transferase M1 (GSTM1) that increases susceptibility to bladder cancer;Bell;J Natl Cancer Inst,1993

5. Metabolic polymorphisms and cancer susceptibility;Smith;Cancer Surv,1995

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