p38 Mitogen-activated protein kinase and alkaline phosphatase in human dental pulp cells
Author:
Publisher
Elsevier BV
Subject
General Dentistry,Otorhinolaryngology,Oral Surgery,Surgery
Reference26 articles.
1. Cells and extracellular matrices of dentin and pulp;Goldberg;Crit Rev Oral Biol Med,2004
2. The role of vascular endothelial growth factor in human dental pulp cells;Matsushita;J Dent Res,2000
3. Proliferation of dental pulp fibroblasts in response to thrombin involves mitogen-activated protein kinase signaling;Gruber;Int Endod J,2004
4. Dental pulp fibroblasts contain target cells for lysophosphatidic Acid;Gruber;J Dent Res,2004
5. Transforming growth factor-beta-1 reduces alkaline phosphatase mRNA and activity and stimulates cell proliferation in cultures of human pulp cells;Shirakawa;J Dent Res,1994
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1. TGFB1/INHBA Homodimer/Nodal-SMAD2/3 Signaling Network: A Pivotal Molecular Target in PDAC Treatment;Molecular Therapy;2021-03
2. Mitogen-activated protein kinase inhibitors reduce the nuclear accumulation of phosphorylated Smads by inhibiting Imp 7 or Imp 8 in HepG2 cells;Oncology Letters;2018-02-02
3. Transforming growth factor beta 1 increases collagen content, and stimulates procollagen I and tissue inhibitor of metalloproteinase-1 production of dental pulp cells: Role of MEK/ERK and activin receptor-like kinase-5/Smad signaling;Journal of the Formosan Medical Association;2017-05
4. Differential effects of p38 and Erk1/2 on the chondrogenic and osteogenic differentiation of dental pulp stem cells;Molecular Medicine Reports;2017-01
5. Signaling Pathways in Dental Stem Cells During Their Maintenance and Differentiation;Dental Stem Cells;2016
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