Muscular degeneration in Duchenne's dystrophy may be caused by a mitochondrial defect
Author:
Publisher
Elsevier BV
Subject
General Medicine
Reference29 articles.
1. Isolation of candidate cDNAs for portions of the Duchenne muscular dystrophy gene;Monaco;Nature,1986
2. Dystrophin: the protein product of the Duchenne muscular dystrophy locus;Hoffman;Cell,1987
3. Ultrastructural localization and the possible role of dystrophin;Harris,1992
4. The fate of dystrophin during the degeneration and regeneration of the soleus muscle of the rat;Vater;Acta Neuropathol,1992
5. The dystrophin connection-ATP?;Bonsett;Med Hypotheses,1992
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Revisiting the dystrophin-ATP connection: How half a century of research still implicates mitochondrial dysfunction in Duchenne Muscular Dystrophy aetiology;Medical Hypotheses;2015-12
2. Evaluation of muscle oxygenation by near-infrared spectroscopy in patients with Becker muscular dystrophy;Neuromuscular Disorders;2012-08
3. Transcriptomic analysis of dystrophin RNAi knockdown reveals a central role for dystrophin in muscle differentiation and contractile apparatus organization;BMC Genomics;2010-06-01
4. A Drug Inhibits the Mitochondrial Protease Inducing Calmitine Deficiency in Skeletal Muscle of Patients with Duchenne's Muscular Dystrophy and dy/dy Dystrophic Mice;Biochemical and Biophysical Research Communications;1997-03
5. Muscle regeneration and mitochondrial calmitine increase in the dystrophic dy/dy mouse after intramuscular chlorpromazine injection;Acta Neuropathologica;1995-09
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