Residual association at C9orf72 suggests an alternative amyotrophic lateral sclerosis-causing hexanucleotide repeat-Reference-Cited by-同舟云学术

Residual association at C9orf72 suggests an alternative amyotrophic lateral sclerosis-causing hexanucleotide repeat

Published:2013-09 Issue:9 Volume:34 Page:2234.e1-2234.e7
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Jones Ashley R.,Woollacott Ione,Shatunov Aleksey,Cooper-Knock Johnathan,Buchman Vladimir,Sproviero William,Smith Bradley,Scott Kirsten M.,Balendra Rubika,Abel Olubunmi,McGuffin Peter,Ellis Catherine M.,Shaw Pamela J.,Morrison Karen E.,Farmer Anne,Lewis Cathryn M.,Leigh P. Nigel,Shaw Christopher E.,Powell John F.,Al-Chalabi Ammar

Funder

Motor Neuron Disease Association of Great Britain and Northern Ireland

European Community's Health Seventh Framework Programme

Welcome Trust

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference37 articles.

1. Deletions of the heavy neurofilament subunit tail in amyotrophic lateral sclerosis;Al-Chalabi;Hum. Mol. Genet.,1999

2. An estimate of amyotrophic lateral sclerosis heritability using twin data;Al-Chalabi;J. Neurol. Neurosurg. Psychiatry,2010

3. Modelling the effects of penetrance and family size on rates of sporadic and familial disease;Al-Chalabi;Hum. Hered.,2011