Lack of RAB39B mutations in early-onset and familial Parkinson's disease in a Taiwanese cohort
Author:
Funder
National Ministry of Science and Technology
Publisher
Elsevier BV
Subject
Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience
Reference19 articles.
1. Mutational analysis of SYNJ1 gene (PARK20) in Parkinson's disease in a Taiwanese population;Chen;Neurobiol. Aging,2015
2. Isolation and characterization of a human novel RAB (RAB39B) gene;Cheng;Cytogenet. Genome Res.,2002
3. Projected number of people with Parkinson disease in the most populous nations, 2005 through 2030;Dorsey;Neurology,2007
4. Lack of CHCHD2 mutations in Parkinson's disease in a Taiwanese population;Fan;Neurobiol. Aging,2016
5. Genetics in Parkinson disease: Mendelian versus non-Mendelian inheritance;Hernandez;J. Neurochem.,2016
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1. Loss of RAB39B does not alter MPTP-induced Parkinson’s disease-like phenotypes in mice;Frontiers in Aging Neuroscience;2023-01-25
2. Dysfunction of RAB39B‐ Mediated Vesicular Trafficking in Lewy Body Diseases;Movement Disorders;2021-05-03
3. RAB39B's role in membrane traffic, autophagy, and associated neuropathology;Journal of Cellular Physiology;2020-08-06
4. Genetic Analysis of RAB39B in an Early-Onset Parkinson's Disease Cohort;Frontiers in Neurology;2020-06-26
5. Genetic analysis of PODXL gene in patients with familial and young-onset Parkinson's disease in a Taiwanese population;Neurobiology of Aging;2019-12
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