Impaired neurogenesis, neuronal loss, and brain functional deficits in the APPxPS1-Ki mouse model of Alzheimer's disease-Reference-Cited by-同舟云学术

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Impaired neurogenesis, neuronal loss, and brain functional deficits in the APPxPS1-Ki mouse model of Alzheimer's disease

Published:2011-03 Issue:3 Volume:32 Page:407-418
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Faure A.,Verret L.,Bozon B.,El Tannir El Tayara N.,Ly M.,Kober F.,Dhenain M.,Rampon C.,Delatour B.

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference46 articles.

1. Multivariate and univariate analysis of continuous arterial spin labeling perfusion MRI in Alzheimer's disease;Asllani;J. Cereb. Blood Flow Metab.,2008

2. Intraneuronal beta-amyloid is a major risk factor—novel evidence from the APP/PS1KI mouse model;Bayer;Neurodegener. Dis.,2008

3. Review on the APP/PS1KI mouse model: intraneuronal Abeta accumulation triggers axonopathy, neuron loss and working memory impairment;Bayer;Genes Brain Behav.,2008

4. Time sequence of maturation of dystrophic neurites associated with Abeta deposits in APP/PS1 transgenic mice;Blanchard;Exp. Neurol.,2003

5. Spatial navigation impairment in mice lacking cerebellar LTD: a motor adaptation deficit?;Burguiere;Nat. Neurosci.,2005

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