Neocortical synaptic bouton number is maintained despite robust amyloid deposition in APP23 transgenic mice-Reference-Cited by-同舟云学术

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Neocortical synaptic bouton number is maintained despite robust amyloid deposition in APP23 transgenic mice

Published:2005-05 Issue:5 Volume:26 Page:607-613
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Boncristiano Sonia,Calhoun Michael E.,Howard Victor,Bondolfi Luca,Kaeser Stephan A.,Wiederhold Karl-Heinz,Staufenbiel Matthias,Jucker Mathias

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference48 articles.

1. Cholinergic changes in the APP23 transgenic mouse model of cerebral amyloidosis;Boncristiano;J Neurosci,2002

2. Amyloid-associated neuron loss and gliogenesis in the neocortex of amyloid precursor protein transgenic mice;Bondolfi;J Neurosci,2002

3. PDAPP; YFP double transgenic mice: a tool to study amyloid-beta associated changes in axonal, dendritic, and synaptic structures;Brendza;J Comp Neurol,2003

4. Modulation of Alzheimer-like synaptic and cholinergic deficits in transgenic mice by human apolipoprotein E depends on isoform, aging, and overexpression of amyloid beta peptides but not on plaque formation;Buttini;J Neurosci,2002

5. Neuronal overexpression of mutant amyloid precursor protein results in prominent deposition of cerebrovascular amyloid;Calhoun;Proc Natl Acad Sci USA,1999

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