Deficient cerebral clearance of vasculotropic mutant Dutch/Iowa Double Aß in human AßPP transgenic mice-Reference-Cited by-同舟云学术

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Deficient cerebral clearance of vasculotropic mutant Dutch/Iowa Double Aß in human AßPP transgenic mice

Published:2006-07 Issue:7 Volume:27 Page:946-954
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Davis Judianne,Xu Feng,Miao Jianting,Previti Mary Lou,Romanov Galina,Ziegler Kelly,Van Nostrand William E.

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference42 articles.

1. Familial Alzheimer's disease-linked presenilin 1 variants elevate Abeta1–42/1–40 ratio in vitro and in vivo;Borchelt;Neuron,1996

2. Early-onset Alzheimer's disease caused by mutations at codon 717 of the beta-amyloid precursor protein gene;Chartier-Harlin;Nature,1991

3. Mutation of the ß-amyloid precursor protein in familial Alzheimer's disease increases ß-protein production;Citron;Nature,1992

4. Enhanced pathologic properties of Dutch-type mutant amyloid ß-protein;Davis;Proc Natl Acad Sci USA,1996

5. Early-onset and robust cerebral microvascular accumulation of amyloid beta-protein in transgenic mice expressing low levels of a vasculotropic Dutch/Iowa mutant form of amyloid ß-protein precursor;Davis;J Biol Chem,2004

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