Transcriptional control of cell cycle-dependent kinase 4 by Smad proteins—implications for Alzheimer's disease-Reference-Cited by-同舟云学术

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Transcriptional control of cell cycle-dependent kinase 4 by Smad proteins—implications for Alzheimer's disease

Published:2012-12 Issue:12 Volume:33 Page:2827-2840
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Ueberham Uwe,Hilbrich Isabel,Ueberham Elke,Rohn Susanne,Glöckner Pia,Dietrich Katharina,Brückner Martina K.,Arendt Thomas

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference89 articles.

1. Cognitive decline in Alzheimer's disease is associated with selective changes in calcineurin/NFAT signaling;Abdul;J. Neurosci,2009

2. Nitric oxide induces TIMP-1 expression by activating the transforming growth factor beta-Smad signaling pathway;Akool;J. Biol. Chem,2005

3. Alzheimer's disease as a loss of differentiation control in a subset of neurons that retain immature features in the adult brain;Arendt;Neurobiol. Aging,2000

4. Synaptic plasticity and cell cycle activation in neurons are alternative effector pathways: the “DrJekyll and MrHyde concept” of Alzheimer's disease or the yin and yang of neuroplasticity;Arendt;Prog. Neurobiol,2003

5. Selective cell death of hyperploid neurons in Alzheimer's disease;Arendt;Am. J. Pathol,2010

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