Enhanced proteasome-dependent degradation of the CDK inhibitor p27kip1 in immortalized lymphocytes from Alzheimer's dementia patients-Reference-Cited by-同舟云学术

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Enhanced proteasome-dependent degradation of the CDK inhibitor p27kip1 in immortalized lymphocytes from Alzheimer's dementia patients

Published:2008-10 Issue:10 Volume:29 Page:1474-1484
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Muñoz Úrsula,Bartolomé Fernando,Bermejo Félix,Martín-Requero Ángeles

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference55 articles.

1. BCR-ABL induces the expression of Skp2 through the PI3K pathway to promote p27Kip1 degradation and proliferation of chronic myelogenous leukemia cells;Andreu;Cancer Res.,2005

2. Synaptic plasticity and cell cycle activation in neurons are alternative effector pathways: the ‘Dr. Jekyll and Mr. Hyde concept’ of Alzheimer's disease or the yin and yang of neuroplasticity;Arendt;Prog. Neurobiol.,2003

3. Peripheral proteasome and caspase activity in Parkinson disease and Alzheimer disease;Blandini;Neurology,2006

4. Deregulated degradation of the cdk inhibitor p27 and malignant transformation;Bloom;Semin. Cancer Biol.,2003

5. A growth factor-dependent nuclear kinase phosphorylates p27(Kip1) and regulates cell cycle progression;Boehm;EMBO J.,2002

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