Iron overload accelerates neuronal amyloid-β production and cognitive impairment in transgenic mice model of Alzheimer's disease-Reference-Cited by-同舟云学术

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Iron overload accelerates neuronal amyloid-β production and cognitive impairment in transgenic mice model of Alzheimer's disease

Published:2014-10 Issue:10 Volume:35 Page:2288-2301
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Becerril-Ortega Javier,Bordji Karim,Fréret Thomas,Rush Travis,Buisson Alain

Funder

Fondation Neurodis

Agence Nationale de la Recherche

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference50 articles.

1. In vivo evaluation of brain iron in Alzheimer disease using magnetic resonance imaging;Bartzokis;Arch. Gen. Psychiatry,2000

2. Activation of extrasynaptic, but not synaptic, NMDA receptors modifies amyloid precursor protein expression pattern and increases amyloid-production;Bordji;J. Neurosci.,2010

3. NMDA receptor-nitric oxide transmission mediates neuronal iron homeostasis via the GTPase Dexras1;Cheah;Neuron,2006

4. HFE mutations and Alzheimer's disease;Connor;J. Alzheimers Dis.,2006

5. Regulation of iron acquisition and storage: consequences for iron-linked disorders;De Domenico;Nat. Rev. Mol. Cell Biol.,2008

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