Superoxide dismutase 1 mutation in a cellular model of amyotrophic lateral sclerosis shifts energy generation from oxidative phosphorylation to glycolysis-Reference-Cited by-同舟云学术

Superoxide dismutase 1 mutation in a cellular model of amyotrophic lateral sclerosis shifts energy generation from oxidative phosphorylation to glycolysis

Published:2014-06 Issue:6 Volume:35 Page:1499-1509
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Allen Scott P.,Rajan Sandeep,Duffy Lynn,Mortiboys Heather,Higginbottom Adrian,Grierson Andrew J.,Shaw Pamela J.

Funder

European Union under the 7th Framework Program for RTD

Project MitoTarget - Grant Agreement

Sheffield Institute for Translational Neuroscience

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference49 articles.

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4. High-fat diet ameliorates neurological deficits caused by defective astrocyte lipid metabolism;Camargo;FASEB J.,2012

5. Expression of a Cu, Zn superoxide dismutase typical of familial amyotrophic lateral sclerosis induces mitochondrial alteration and increase of cytosolic Ca2+ concentration in transfected neuroblastoma SH-SY5Y cells;Carri;FEBS Lett.,1997

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