Effect of long-term treatment with rasagiline on cognitive deficits and related molecular cascades in aged mice-Reference-Cited by-同舟云学术

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Effect of long-term treatment with rasagiline on cognitive deficits and related molecular cascades in aged mice

Published:2015-09 Issue:9 Volume:36 Page:2628-2636
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Weinreb Orly,Badinter Felix,Amit Tamar,Bar-Am Orit,Youdim Moussa B.H.

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference57 articles.

1. Mitochondrial permeability transition mediates apoptosis induced by N- methyl(R)salsolinol, an endogenous neurotoxin, and is inhibited by Bcl- 2 and rasagiline, N-propargyl-1(R)-aminoindan;Akao;J. Neurochem.,2002

2. An anti-Parkinson's disease drug, N-propargyl-1(R)-aminoindan (rasagiline), enhances expression of anti-apoptotic Bcl-2 in human dopaminergic SH-SY5Y cells;Akao;Neurosci. Lett.,2002

3. Regulation of Bcl-2 family proteins, neurotrophic factors, and APP processing in the neurorescue activity of propargylamine;Bar-Am;FASEB J.,2005

4. The novel cholinesterase-monoamine oxidase inhibitor and antioxidant, ladostigil, confers neuroprotection in neuroblastoma cells and aged rats;Bar-Am;J. Mol. Neurosci.,2009

5. The neuroprotective mechanism of 1-(R)-aminoindan, the major metabolite of the anti-parkinsonian drug rasagiline;Bar-Am;J. Neurochem.,2010

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