Early neuronal dysfunction by amyloid β oligomers depends on activation of NR2B-containing NMDA receptors-Reference-Cited by-同舟云学术

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Early neuronal dysfunction by amyloid β oligomers depends on activation of NR2B-containing NMDA receptors

Published:2011-12 Issue:12 Volume:32 Page:2219-2228
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Rönicke Raik,Mikhaylova Marina,Rönicke Sabine,Meinhardt Jessica,Schröder Ulrich H.,Fändrich Marcus,Reiser Georg,Kreutz Michael R.,Reymann Klaus G.

Funder

BMBF (BioFuture) and DFG

DFG

Schram foundation

Exzellenznetzwerk Biowissenschaften

EFRE ZVOH

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference37 articles.

1. The role of NMDAR subtypes and charge transfer during hippocampal LTP induction;Berberich;Neuropharmacology,2007

2. Clusters of hyperactive neurons near amyloid plaques in a mouse model of Alzheimer's disease;Busche;Science,2008

3. Rapid, concurrent alterations in pre- and postsynaptic structure induced by naturally-secreted amyloid-beta protein;Calabrese;Mol. Cell. Neurosci.,2007

4. Reduction of the threshold for long-term potentiation by prior theta-frequency synaptic activity;Christie;Hippocampus,1995

5. Determinants of spontaneous activity in networks of cultured hippocampus;Cohen;Brain Res.,2008

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