Age- and tissue-specific changes in mitochondrial and nuclear DNA base excision repair activity in mice: Susceptibility of skeletal muscles to oxidative injury-Reference-Cited by-同舟云学术

Age- and tissue-specific changes in mitochondrial and nuclear DNA base excision repair activity in mice: Susceptibility of skeletal muscles to oxidative injury

Published:2010-05 Issue:5 Volume:131 Page:330-337
ISSN:0047-6374
Container-title:Mechanisms of Ageing and Development
language:en
Short-container-title:Mechanisms of Ageing and Development

Author:

Szczesny Bartosz,Tann Anne W.,Mitra Sankar

Publisher

Elsevier BV

Subject

Developmental Biology,Ageing

Reference53 articles.

1. Repair of U/G and U/A in DNA by UNG2-associated repair complexes takes place predominantly by short-patch repair both in proliferating and growth-arrested cells;Akbari;Nucleic Acids Res.,2004

2. Mitochondrial base excision repair of uracil and AP sites takes place by single-nucleotide insertion and long-patch DNA synthesis;Akbari;DNA Repair (Amst.),2008

3. A unified view of base excision repair: lesion-dependent protein complexes regulated by post-translational modification;Almeida;DNA Repair (Amst.),2007

4. Attenuation of DNA polymerase beta-dependent base excision repair and increased DMS-induced mutagenicity in aged mice;Cabelof;Mutat. Res.,2002

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