Genetic deletion of TRPA1 receptor attenuates amyloid beta- 1-42 (Aβ1-42)-induced neurotoxicity in the mouse basal forebrain in vivo-Reference-Cited by-同舟云学术

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Genetic deletion of TRPA1 receptor attenuates amyloid beta- 1-42 (Aβ1-42)-induced neurotoxicity in the mouse basal forebrain in vivo

Published:2020-07 Issue: Volume:189 Page:111268
ISSN:0047-6374
Container-title:Mechanisms of Ageing and Development
language:en
Short-container-title:Mechanisms of Ageing and Development

Author:

Payrits M.,Borbely E.,Godo S.,Ernszt D.,Kemeny A.,Kardos J.,Szoke E.,Pinter E.

Funder

Hungarian Scientific Research Fund

Richter Gedeon Talentum Alapítvány

Publisher

Elsevier BV

Subject

Developmental Biology,Ageing

Reference42 articles.

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2. Object recognition in rats and mice: A one-trial non-matching-to-sample learning task to study ‘recognition memory’;Bevins;Nat. Protoc.,2006

3. Behavioural alterations and morphological changes are attenuated by the lack of TRPA1 receptors in the cuprizone-induced demyelination model in mice;Bölcskei;J. Neuroimmunol.,2018

4. TRPA1 channels promote astrocytic Ca2+ hyperactivity and synaptic dysfunction mediated by oligomeric forms of amyloid-β peptide;Bosson;Mol. Neurodegener.,2017

5. Polymorphisms of pro-inflammatory genes and Alzheimer’s disease risk: a pharmacogenomic approach;Candore;Mech. Ageing Dev.,2007

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