Terminal complement complex C5b-9 reduced megalin and cubilin-mediated tubule proteins uptake in a mouse model of trichloroethylene hypersensitivity syndrome-Reference-Cited by-同舟云学术

Terminal complement complex C5b-9 reduced megalin and cubilin-mediated tubule proteins uptake in a mouse model of trichloroethylene hypersensitivity syndrome

Published:2019-12 Issue: Volume:317 Page:110-119
ISSN:0378-4274
Container-title:Toxicology Letters
language:en
Short-container-title:Toxicology Letters

Author:

Wang Feng^ORCID,Huang Li-ping^ORCID,Dai Yu-ying,Huang Meng,Jiang Wei,Ye Liang-ping,Zhu Qi-xing

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference39 articles.

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4. Cadmium complexed with beta2-Microglubulin, albumin and Lipocalin-2 rather than metallothionein cause megalin: cubilin dependent toxicity of the renal proximal tubule;Fels;Int. J. Mol. Sci.,2019

5. Historical occupational trichloroethylene air concentrations based on inspection measurements from Shanghai, China;Friesen;Ann. Occup. Hyg.,2015

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1. Trichloroethylene exposure, multi-organ injury, and potential mechanisms: A narrative review;Science of The Total Environment;2024-10

2. C5b-9 promotes ferritinophagy leading to ferroptosis in renal tubular epithelial cells of trichloroethylene-sensitized mice;Science of The Total Environment;2024-05

3. IP3R-dependent mitochondrial dysfunction mediates C5b-9-induced ferroptosis in trichloroethylene-caused immune kidney injury;Frontiers in Immunology;2023-06-13

4. Trichloroethylene induces immune renal tubular injury through SIRT 1/HSP 70/TLR 4 pathway in BALBc mice;International Immunopharmacology;2022-11

5. C5b-9 mediates ferroptosis of tubular epithelial cells in trichloroethylene-sensitization mice;Ecotoxicology and Environmental Safety;2022-10