Behavioral deficits in the cuprizone-induced murine model of demyelination/remyelination
Author:
Publisher
Elsevier BV
Subject
Toxicology,General Medicine
Reference29 articles.
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1. Preventing production of new oligodendrocytes impairs remyelination and sustains behavioural deficits after demyelination;Biochemical and Biophysical Research Communications;2024-11
2. Lactoferrin, chitosan double-coated oleosomes loaded with clobetasol propionate for remyelination in multiple sclerosis: Physicochemical characterization and in-vivo assessment in a cuprizone-induced demyelination model.;International Journal of Biological Macromolecules;2024-10
3. Targeting the PAC1 receptor mitigates degradation of myelin and synaptic markers and diminishes locomotor deficits in the cuprizone demyelination model;Journal of Neurochemistry;2024-08-08
4. Transient demyelination causes long‐term cognitive impairment, myelin alteration and network synchrony defects;Glia;2024-02-16
5. Evidence for decreased copper associated with demyelination in the corpus callosum of cuprizone-treated mice;Metallomics;2024-01
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