Variable p53/Nrf2 crosstalk contributes to triptolide-induced hepatotoxic process-Reference-Cited by-同舟云学术

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Variable p53/Nrf2 crosstalk contributes to triptolide-induced hepatotoxic process

Published:2023-04 Issue: Volume:379 Page:67-75
ISSN:0378-4274
Container-title:Toxicology Letters
language:en
Short-container-title:Toxicology Letters

Author:

Hou Zhenyan,Yan Miao,Li Huixiang,Wang Weida,You Shen,Wang Mingjin,Du Tingting,Gong Hui,Li Wenqun,Guo Lin,Wei Shanshan,Zhang Bikui,Ji Ming,Chen Xiaoguang

Funder

Chinese Academy of Meteorological Sciences

CAMS Innovation Fund for Medical Sciences

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference37 articles.

1. A mechanism of ubiquitin-independent proteasomal degradation of the tumor suppressors p53 and p73;Asher;Genes Dev.,2005

2. Mdm-2 and ubiquitin-independent p53 proteasomal degradation regulated by NQO1;Asherm;Proc. Natl. Acad. Sci. USA,2002

3. Regulation of expression of drug-metabolizing enzymes by oncogenic signaling pathways in liver tumors: a review;Braeuning;Acta Pharm. Sin. B,2019

4. The ethanol extract of licorice (Glycyrrhiza uralensis) protects against triptolide-induced oxidative stress through activation of Nrf2;Cao;Evid. Based Complement. Altern. Med.,2017

5. NRF2 is a major target of ARF in p53-independent tumor suppression;Chen;Mol. Cell,2017

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Recent advances in the pharmacological applications and liver toxicity of triptolide;Chemico-Biological Interactions;2023-09

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