Acute mercury toxicity modulates cytochrome P450, soluble epoxide hydrolase and their associated arachidonic acid metabolites in C57Bl/6 mouse heart-Reference-Cited by-同舟云学术

Acute mercury toxicity modulates cytochrome P450, soluble epoxide hydrolase and their associated arachidonic acid metabolites in C57Bl/6 mouse heart

Published:2014-04 Issue:1 Volume:226 Page:53-62
ISSN:0378-4274
Container-title:Toxicology Letters
language:en
Short-container-title:Toxicology Letters

Author:

Amara Issa E.A.,Elshenawy Osama H.,Abdelrady Mohamed,El-Kadi Ayman O.S.

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference54 articles.

1. Inhibition of soluble epoxide hydrolase confers cardioprotection and prevents cardiac cytochrome P450 induction by benzo(a)pyrene;Aboutabl;J. Cardiovasc. Pharmacol.,2011

2. Mercury induces proliferation and reduces cell size in vascular smooth muscle cells through MAPK, oxidative stress and cyclooxygenase-2 pathways;Aguado;Toxicol. Appl. Pharmacol.,2013

3. Soluble epoxide hydrolase plays an essential role in angiotensin II-induced cardiac hypertrophy;Ai;PNAS,2009

4. Fenofibrate modulates cytochrome P450 and arachidonic acid metabolism in the heart and protects against isoproterenol-induced cardiac hypertrophy;Althurwi;J. Cardiovasc. Pharmacol.,2013

5. Soluble epoxide hydrolase inhibitor, TUPS, protects against isoprenaline-induced cardiac hypertrophy;Althurwi;Br. J. Pharmacol.,2013

Cited by 23 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Mercury-induced toxicity: Mechanisms, molecular pathways, and gene regulation;Science of The Total Environment;2024-09

3. Interaction of mercury species with proteins: towards possible mechanism of mercurial toxicology;Toxicology Research;2023-05-30

4. Mercury Induced Tissue Damage, Redox Metabolism, Ion Transport, Apoptosis, and Intestinal Microbiota Change in Red Swamp Crayfish (Procambarus clarkii): Application of Multi-Omics Analysis in Risk Assessment of Hg;Antioxidants;2022-09-29

5. Urinary metals as influencing factors of coronary heart disease among a population in Guangzhou, China;Ecotoxicology and Environmental Safety;2022-08