Down-regulation of O-GlcNAcylation alleviates insulin signaling pathway impairment following arsenic exposure via suppressing the AMPK/mTOR-autophagy pathway

Author:

Zhang Wenxin,Zeng Shuxian,Huang Jieliang,Tian Xianbing,Wu Jiegen,Guo Lianxian,Liang Yi

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Reference52 articles.

1. Arsenic uptake, toxicity, detoxification, and speciation in plants: physiological, biochemical, and molecular aspects;Abbas;Int J. Environ. Res Public Health,2018

2. Arsenic exposure and type 2 diabetes: micrornas as mechanistic links?;Beck;Curr. Diab Rep.,2017

3. LC/MS/MS-Based Liver metabolomics to identify chronic liver injury biomarkers following exposure to arsenic in rats;Bi;Biol. Trace Elem. Res,2022

4. Role of SESTRIN2/AMPK/ULK1 pathway activation and lysosomes dysfunction in NaAsO(2)-induced liver injury under oxidative stress';Bi;Ecotoxicol. Environ. Saf.,2023

5. Nrf2 and HIF1alpha converge to arsenic-induced metabolic reprogramming and the formation of the cancer stem-like cells;Bi;Theranostics,2020

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