Comparison of 2,2-bis(bromomethyl)-1,3-propanediol induced genotoxicity in UROtsa cells and primary rat hepatocytes: Relevance of metabolism and oxidative stress-Reference-Cited by-同舟云学术

Comparison of 2,2-bis(bromomethyl)-1,3-propanediol induced genotoxicity in UROtsa cells and primary rat hepatocytes: Relevance of metabolism and oxidative stress

Published:2013-10 Issue:3 Volume:222 Page:273-279
ISSN:0378-4274
Container-title:Toxicology Letters
language:en
Short-container-title:Toxicology Letters

Author:

Kong Weixi,Gu Pengfei,Knudsen Gabriel A.,Sipes I. Glenn

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

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1. Cellular and physiological mechanisms of halogenated and organophosphorus flame retardant toxicity;Science of The Total Environment;2023-11

2. Oxidative debromination of 2,2-bis(bromomethyl)-1,3-propanediol by UV/persulfate process and corresponding formation of brominated by-products;Chemosphere;2019-08

3. Functional genomic assessment of 2, 2-bis (bromomethyl)-1, 3-propanediol induced cytotoxicity in a single-gene knockout library of E. coli;Chemosphere;2017-10