Cerebral formation in situ of S-carboxymethylcysteine after ifosfamide administration to mice: A further clue to the mechanism of ifosfamide encephalopathy

Author:

Lerch Stefanie,Küpfer Adrian,Idle Jeffrey R.,Lauterburg Bernhard H.

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference32 articles.

1. Inhibition of (mono)amine oxidase activity and prevention of ifosfamide encephalopathy by methylene blue;Aeschlimann;Drug Metab. Dispos.,1996

2. Quantification of S-carboxymethyl-(R)-cysteine in human plasma by high-performance ion-exchange liquid chromatography/atmospheric pressure ionization mass spectrometry;Anacardio;J. Mass Spectrom.,1997

3. Comparison of continuous infusion and bolus administration of ifosfamide in children;Boddy;Eur. J. Cancer,1995

4. Insights into the mechanisms of ifosfamide encephalopathy: drug metabolites have agonistic effects on alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)/kainate receptors and induce cellular acidification in mouse cortical neurons;Chatton;J. Pharmacol. Exp. Ther.,2001

5. Monoamine oxidase and cerebral uptake of dopaminergic drugs;Cotzias;Proc. Natl. Acad. Sci. U.S.A.,1974

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