Altered release of thrombomodulin and HMGB1 in the placenta complicated with preeclampsia
Author:
Funder
Japan Society for the Promotion of Science
Publisher
Elsevier BV
Subject
Developmental Biology,Obstetrics and Gynecology,Reproductive Medicine
Reference28 articles.
1. Pathogenesis and genetics of pre-eclampsia;Roberts;Lancet,2001
2. Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia;Maynard;J. Clin. Invest.,2003
3. Thrombomodulin promotes placental function by up-regulating placental growth factor via inhibition of high-mobility-group box 1 and hypoxia-inducible factor 1alpha;Oda;Placenta,2021
4. Thrombomodulin and pregnancy in the limelight: insights into the therapeutic aspect of thrombomodulin in pregnancy complications;Oda;J. Thromb. Haemostasis,2022
5. Thrombomodulin is found on endothelium of arteries, veins, capillaries, and lymphatics, and on syncytiotrophoblast of human placenta;Maruyama;J. Cell Biol.,1985
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