Prenatal caffeine exposure induced high susceptibility to metabolic syndrome in adult female offspring rats and its underlying mechanisms-Reference-Cited by-同舟云学术

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Prenatal caffeine exposure induced high susceptibility to metabolic syndrome in adult female offspring rats and its underlying mechanisms

Published:2017-08 Issue: Volume:71 Page:150-158
ISSN:0890-6238
Container-title:Reproductive Toxicology
language:en
Short-container-title:Reproductive Toxicology

Author:

Pei Lin-guo,Yuan Chao,Guo Yi-tian,Kou Hao,Xia Li-ping,Zhang Li,Yan You-e,Xu Dan,Wang Hui

Funder

National Natural Science Foundation of China

Hubei Province Health and Family Planning Scientific Research Project

Young Investigator Program of Zhongnan Hospital of Wuhan University

Publisher

Elsevier BV

Subject

Toxicology

Reference55 articles.

1. The metabolic syndrome: common origins of a multifactorial disorder;Bruce;Postgrad. Med. J.,2009

2. The metabolic syndrome;Eckel;Lancet,2010

3. Adipokines and insulin resistance;Rabe;Mol. Med.,2008

4. Intra-uterine growth retardation as a risk factor of postnatal metabolic disorders;Mierzynski;Curr. Pharm. Biotechnol.,2016

5. In utero and neonate exposure to nonylphenol develops hyperadrenalism and metabolic syndrome later in life. I. First generation rats (F(1));Chang;Toxicology,2012

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1. Association between Coffee Consumption, Caffeine Intake, and Metabolic Syndrome Severity in Patients with Self-Reported Rheumatoid Arthritis: National Health and Nutrition Examination Survey 2003–2018;Nutrients;2022-12-26

2. P-gp expression inhibition mediates placental glucocorticoid barrier opening and fetal weight loss;BMC Medicine;2021-12

3. The low‐expression programming of 11β‐HSD2 mediates osteoporosis susceptibility induced by prenatal caffeine exposure in male offspring rats;British Journal of Pharmacology;2020-08-20

4. Molecular mechanisms governing offspring metabolic programming in rodent models of in utero stress;Cellular and Molecular Life Sciences;2020-06-03

5. Prenatal ethanol exposure induced disorder of hypothalamic-pituitary-adrenal axis-associated neuroendocrine metabolic programming alteration and dysfunction of glucose and lipid metabolism in 40-week-old female offspring rats;Reproductive Toxicology;2020-06

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