Glucose-stimulated increase in cytoplasmic pH precedes increase in free Ca2+ in pancreatic beta-cells. A possible role for pyruvate.
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference50 articles.
1. Ca2+, cAMP, and phospholipid-derived messengers in coupling mechanisms of insulin secretion
2. Pancreatic islet glucose metabolism and regulation of insulin secretion
3. Oscillations in cytosolic free Ca2+, oxygen consumption, and insulin secretion in glucose-stimulated rat pancreatic islets
4. Inhibition of ATP-regulated K+ channels precedes depolarization-induced increase in cytoplasmic free Ca2+ concentration in pancreatic beta-cells.
5. Glucose-induced increase in cytoplasmic pH in pancreatic beta-cells is mediated by Na+/H+ exchange, an effect not dependent on protein kinase C.
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1. Pyruvate Kinase Controls Signal Strength in the Insulin Secretory Pathway;Cell Metabolism;2020-11
2. Molecular Regulations and Functions of the Transient Receptor Potential Channels of the Islets of Langerhans and Insulinoma Cells;Cells;2020-03-11
3. Mitochondrial Metabolism of Pyruvate Is Essential for Regulating Glucose-stimulated Insulin Secretion;Journal of Biological Chemistry;2014-05
4. Diphenylhydantoin Suppresses Glucose-Induced Insulin Release by Decreasing Cytoplasmic H+ Concentration in Pancreatic Islets;Endocrinology;2006-06-01
5. Dihydroxyacetone-induced Oscillations in Cytoplasmic Free Ca2+ and the ATP/ADP Ratio in Pancreatic β-Cells at Substimulatory Glucose;Journal of Biological Chemistry;2003-10
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