Lack of coupling of D-2 receptors to adenylate cyclase in GH-3 cells exposed to epidermal growth factor. Possible role of a differential expression of Gi protein subtypes.-Reference-Cited by-同舟云学术

Lack of coupling of D-2 receptors to adenylate cyclase in GH-3 cells exposed to epidermal growth factor. Possible role of a differential expression of Gi protein subtypes.

Published:1991-12 Issue:34 Volume:266 Page:23392-23398
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Missale C.,Boroni F.,Castelletti L.,Dal Toso R.,Gabellini N.,Sigala S.,Spano P.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference49 articles.

1. PRODUCTION OF BOTH PROLACTIN AND GROWTH HORMONE BY CLONAL STRAINS OF RAT PITUITARY TUMOR CELLS

2. Establishment of Clonal Strains of Rat Pituitary Tumor Cells That Secrete Growth Hormone1,2

3. Inhibition of Prolactin Secretion and Synthesis by Dopamine, Noradrenaline and Pilocarpine in Cultured Rat Pituitary Tumour Cells

4. Defective dopaminergic regulation of prolactin secretion in a rat pituitary tumour cell line

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3. Epidermal Growth Factor Promotes Uncoupling from Adenylyl Cyclase of the Rat D2S Receptor Expressed in GH4C1 Cells;Journal of Neurochemistry;2008-06-28

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