Evidence that an approximately 50-kDa mammalian plasma membrane protein with receptor-like properties mediates the amphiphilicity of specifically bound Clostridium perfringens enterotoxin.-Reference-Cited by-同舟云学术

Evidence that an approximately 50-kDa mammalian plasma membrane protein with receptor-like properties mediates the amphiphilicity of specifically bound Clostridium perfringens enterotoxin.

Published:1994-04 Issue:14 Volume:269 Page:10838-10848
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Wieckowski E.U.,Wnek A.P.,McClane B.A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference45 articles.

1. Clostridium perfringens enterotoxin acts by producing small molecule permeability alterations in plasma membranes

2. Clostridium perfringens enterotoxin

3. Pharmacology of Bacterial. Toxins;McDonel,1986

4. The relationship between cytotoxic effect and binding to mammalian cultured cells ofClostridium perfringensenterotoxin

5. Osmotic stabilizers differentially inhibit permeability alterations induced in vero cells by Clostridium Perfringens enterotoxin

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