Peripheral involvement of PKA and PKC in subcutaneous bee venom-induced persistent nociception, mechanical hyperalgesia, and inflammation in rats-Reference-Cited by-同舟云学术

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Peripheral involvement of PKA and PKC in subcutaneous bee venom-induced persistent nociception, mechanical hyperalgesia, and inflammation in rats

Published:2008-03 Issue:1 Volume:135 Page:31-36
ISSN:0304-3959
Container-title:Pain
language:en
Short-container-title:

Author:

Chen Hui-Sheng,Lei Jing,He Xiang,Qu Fang,Wang Yang,Wen Wei-Wei,You Hao-Jun,Arendt-Nielsen Lars

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Anesthesiology and Pain Medicine,Clinical Neurology,Neurology

Reference42 articles.

1. Protein kinase C mediates up-regulation of tetrodotoxin-resistant, persistent Na current in rat and mouse sensory neurons.;Baker;J Physiol,2005

2. Constitutive and inducible mechanisms for synthesis and release of cytokines in immune cell lines.;Baumgartner;J Immunol,1996

3. cAMP-dependent protein kinase regulates desensitization of the capsaicin receptor (VR1) by direct phosphorylation.;Bhave;Neuron,2002

4. Secondary heat, but not mechanical, hyperalgesia induced by subcutaneous injection of bee venom in the conscious rat: effect of systemic MK-801, a non-competitive NMDA receptor antagonist.;Chen;Eur J Pain,2000

5. Contralateral heat hyperalgesia induced by unilaterally intraplantar bee venom injection is produced by central changes: a behavioral study in the conscious rat.;Chen;Neurosci Lett,2000

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