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Activation of the spinal ERK signaling pathway contributes naloxone-precipitated withdrawal in morphine-dependent rats

  • Published:2005-12 Issue:3 Volume:118 Page:336-349
  • ISSN:0304-3959
  • Container-title:Pain
  • language:en
  • Short-container-title:

Author:

Cao Jun-Li,He Jian-Hua,Ding Hai-Lei,Zeng Yin-Ming

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Anesthesiology and Pain Medicine,Clinical Neurology,Neurology

Reference86 articles.

1. The A-type potassium channel KV4.2 is a substrate for the mitogen-activated protein kinase ERK.;Adams;J Neurochem,2000

2. Molecular psychology: roles for the ERK MAP kinase cascade in memory.;Adams;Annu Rev Pharmacol Toxicol,2002

3. Inflammation persistently enhances nociceptive behaviors mediated by spinal group I mGluRs through sustained ERK activation.;Adwanikara;Pain,2004

4. MAP kinase activation by mu opioid receptor involves phosphatidylinositol 3-kinase but not the cAMPPKA pathway.;Ai;Fed Eur Biochem Soc Lett,1999

5. Opioid modulation of extracellular signal-regulated protein kinase activity is ras-dependent and involves G subunits.;Belcheva;J Neurochem,1998
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