High PI3K/mTOR and low MAPK/JNK activity results in decreased apoptosis and autophagy in nasal polyposis

Author:

Simsek FatmaORCID,Eren ErdemORCID,Bahceci SelenORCID,Aladag Ibrahim

Funder

Izmir Katip Çelebi University Scientific Research and Project Coordinatorship

Publisher

Elsevier BV

Subject

Otorhinolaryngology

Reference31 articles.

1. The activity of N-acetyl-β-d-hexosaminidase A and B and β-glucuronidase in nasal polyps and hypertrophic nasal concha;Chojnowska;Otolaryngol Pol,2014

2. Dexamethasone induces apoptosis of nasal polyp-derived tissue cultures through JNK and p38 MAPK activation;Lee;Clin Exp Otorhinolaryngol,2014

3. Autophagy is deficient in nasal polyps: implications for the pathogenesis of the disease;Chen;Int Forum Allergy Rhinol,2015

4. Nasal polyps: origin, etiology, pathogenesis, and structure;Tos,2001

5. The influence of protein kinase C inhibitor in eosinophil apoptosis of nasal polyps;Li;Zhonghua Er Bi Yan Hou Ke Za Zhi,2004

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