Deletion of indoleamine 2,3 dioxygenase (Ido)1 but not Ido2 exacerbates disease symptoms of MOG35-55-induced experimental autoimmune encephalomyelitis-Reference-Cited by-同舟云学术

Deletion of indoleamine 2,3 dioxygenase (Ido)1 but not Ido2 exacerbates disease symptoms of MOG35-55-induced experimental autoimmune encephalomyelitis

Published:2020-08 Issue: Volume:7 Page:100116
ISSN:2666-3546
Container-title:Brain, Behavior, & Immunity - Health
language:en
Short-container-title:Brain, Behavior, & Immunity - Health

Author:

Wetzel Lisa A.,Hurtado Myrna,MacDowell Kaswan Zoe A.,McCusker Robert H.,Steelman Andrew J.

Funder

National Multiple Sclerosis Society

National Institute of Neurological Disorders and Stroke

National Institute of Food and Agriculture

Publisher

Elsevier BV

Subject

General Medicine

Reference46 articles.

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3. Distinct roles of immunoreceptor tyrosine-based motifs in immunosuppressive indoleamine 2,3-dioxygenase 1;Albini;J. Cell Mol. Med.,2017

4. Identification of a 2-propanol analogue modulating the non-enzymatic function of indoleamine 2,3-dioxygenase 1;Albini;Biochem. Pharmacol.,2018

5. Indoleamine 2,3-dioxygenase expression is restricted to fetal trophoblast giant cells during murine gestation and is maternal genome specific;Baban;J. Reprod. Immunol.,2004

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