Complementation of V(D)J Recombination Deficiency in RAG-1−/− B Cells Reveals a Requirement for Novel Elements in the N-Terminus of RAG-1-Reference-Cited by-同舟云学术

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Complementation of V(D)J Recombination Deficiency in RAG-1−/− B Cells Reveals a Requirement for Novel Elements in the N-Terminus of RAG-1

Published:1997-07 Issue:1 Volume:7 Page:13-24
ISSN:1074-7613
Container-title:Immunity
language:en
Short-container-title:Immunity

Author:

Roman Christopher A.J,Cherry Sara R,Baltimore David

Publisher

Elsevier BV

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference70 articles.

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5. Bacteriophage Mu;Baker;Virology,1995

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1. RAG genomic variation causes autoimmune diseases through specific structure-based mechanisms of enzyme dysregulation;iScience;2023-10

2. Nucleolar localization of RAG1 modulates V(D)J recombination activity;Proceedings of the National Academy of Sciences;2020-02-11

3. VprBP (DCAF1) Regulates RAG1 Expression Independently of Dicer by Mediating RAG1 Degradation;The Journal of Immunology;2018-06-20

4. New insights into the evolutionary origins of the recombination‐activating gene proteins and V(D)J recombination;The FEBS Journal;2017-01-06

5. Riches in RAGs: Revealing the V(D)J Recombinase through High-Resolution Structures;Trends in Biochemical Sciences;2017-01

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