Radiation-sensitive irs mutants rejoin DNA double-strand breaks with efficiency similar to that of parental V79 cells but show altered response to radiation-induced G2 delay-Reference-Cited by-同舟云学术

Radiation-sensitive irs mutants rejoin DNA double-strand breaks with efficiency similar to that of parental V79 cells but show altered response to radiation-induced G2 delay

Published:1992-08 Issue:2 Volume:274 Page:111-122
ISSN:0921-8777
Container-title:Mutation Research/DNA Repair
language:en
Short-container-title:Mutation Research/DNA Repair

Author:

Cheong Nge,Wang Ya,Jackson Mary,Iliakis George

Publisher

Elsevier BV

Subject

Genetics,Toxicology,Molecular Biology

Reference29 articles.

1. Vector-mediated DNA double-strand break repair analysis in normal, and radiation-sensitive. Chinese hamster V79 cells;Debenham;Mutation Res.,1988

2. The effects of ionizing radiation on cell cycle progression in ataxia telangiectasia;Ford;Mutation Res.,1984

3. Radiation-induced potentially lethal damage: DNA lesions susceptible to fixation;Iliakis;Int. J. Radiat. Biol.,1988

4. The role of DNA double strand breaks in ionizing radiation induced killing of eucaryotic cells;Iliakis;Bioessays.,1991

5. Radiosensitive xrs-5 and parental CHO cells show identical DNA neutral filter elution dose response: implications for a relationship between cell radiosensitivity and induction of DNA double strand breaks;Iliakis;Int. J. Radiat. Biol.,1988

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