Nuclease modification in Chinese hamster cells hypersensitive to DNA cross-linking agents — A model for Fanconi anemia
Author:
Publisher
Elsevier BV
Subject
Genetics,Toxicology,Molecular Biology
Reference27 articles.
1. The Chinese hamster cell mutant V-H4 is homologous to Fanconi anemia (complementary group A);Arwert;Cytogenet. Cell Genet.,1991
2. Diepoxybutane test for prenatal and postnatal diagnosis of Fanconi anemia;Auerbach,1989
3. Third-chromosome mutagen-sensitive mutants of Drosphila melanogaster;Boyd;Genetics,1981
4. The mus308 mutants of Drosophila exhibit hypersensitivity to DNA cross-linking agents and are defective in a deoxyribonuclease;Boyd;Genetics,1990
5. Abnormal recovery of DNA replication in ultraviolet-irradiated cell cultures of Drosophila melanogaster which are defective in DNA repair;Brown;Mol. Gen. Genet.,1981
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2. The Drosophila mus308 gene product, implicated in tolerance of DNA interstrand crosslinks, is a nuclear protein found in both ovaries and embryos;DNA Repair;2005-08
3. Spatio-temporal expression of Drosophila mitochondrial transcription factor A during development;Cell Biology International;2003-04
4. Mutation: Somatic Mutation, Cancer, and Aging;Human Genetics;1997
5. Spectrum of spontaneously occurring mutations in the HPRT gene of the Chinese hamster V79 cell mutant V-H4, which is homologous to Fanconi anemia group A;Mutagenesis;1996
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