Upregulation of sestrin 2 expression via JNK pathway activation contributes to autophagy induction in cancer cells-Reference-Cited by-同舟云学术

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Upregulation of sestrin 2 expression via JNK pathway activation contributes to autophagy induction in cancer cells

Published:2013-01 Issue:1 Volume:25 Page:150-158
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Zhang Xiao-Yue,Wu Xiao-Qi,Deng Rong,Sun Ting,Feng Gong-Kan,Zhu Xiao-Feng

Funder

National Basic Research Program (973 Program) of China

Major Science and Technology Project of the National Basic Research Program of China

Natural Science Foundation of China

Natural Science Foundation of Guangdong in China

Publisher

Elsevier BV

Subject

Cell Biology

Reference47 articles.

1. Identification of a novel stress-responsive gene Hi95 involved in regulation of cell viability

2. PA26, a novel target of the p53 tumor suppressor and member of the GADD family of DNA damage and growth arrest inducible genes

3. Gadd153 Sensitizes Cells to Endoplasmic Reticulum Stress by Down-Regulating Bcl2 and Perturbing the Cellular Redox State

4. Role of Gadd45 in apoptosis

5. A Family of Stress-Inducible GADD45-like Proteins Mediate Activation of the Stress-Responsive MTK1/MEKK4 MAPKKK

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4. DNMT3a-mediated upregulation of the stress inducible protein sestrin-2 contributes to malignant transformation of human bronchial epithelial cells following nickel exposure;Ecotoxicology and Environmental Safety;2024-02

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