Tamoxifen-induced activation of p21Waf1/Cip1 gene transcription is mediated by Early Growth Response-1 protein through the JNK and p38 MAP kinase/Elk-1 cascades in MDA-MB-361 breast carcinoma cells-Reference-Cited by-同舟云学术

Tamoxifen-induced activation of p21Waf1/Cip1 gene transcription is mediated by Early Growth Response-1 protein through the JNK and p38 MAP kinase/Elk-1 cascades in MDA-MB-361 breast carcinoma cells

Published:2007-06 Issue:6 Volume:19 Page:1290-1300
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Kim Chang Gun,Choi Byeong Hyeok,Son Sang Wook,Yi Seong Joon,Shin Soon Young,Lee Young Han

Publisher

Elsevier BV

Subject

Cell Biology

Reference65 articles.

1. Sex-specific regulation of growth plate chondrocytes by estrogen is via multiple MAP kinase signaling pathways

2. Production and Actions of Estrogens

3. Steroid Hormone Receptor Signaling in Cancer

4. Estrogen Regulation of Cyclin D1 Gene Expression in ZR-75 Breast Cancer Cells Involves Multiple Enhancer Elements

5. Down-regulation of p21WAF1/CIP1 or p27Kip1 abrogates antiestrogen-mediated cell cycle arrest in human breast cancer cells

Cited by 31 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Decoding cell death signalling: Impact on the response of breast cancer cells to approved therapies;Life Sciences;2024-04

2. N6-methyladenosine reader YTHDF2 promotes multiple myeloma cell proliferation through EGR1/p21cip1/waf1/CDK2-Cyclin E1 axis-mediated cell cycle transition;Oncogene;2023-04-03

3. Asprosin inhibits macrophage lipid accumulation and reduces atherosclerotic burden by up-regulating ABCA1 and ABCG1 expression via the p38/Elk-1 pathway;Journal of Translational Medicine;2022-07-28

4. The lncRNA SLNCR Recruits the Androgen Receptor to EGR1-Bound Genes in Melanoma and Inhibits Expression of Tumor Suppressor p21;Cell Reports;2019-05

5. Activation of MET promotes resistance to sorafenib in hepatocellular carcinoma cells via the AKT/ERK1/2-EGR1 pathway;Artificial Cells, Nanomedicine, and Biotechnology;2019-01-19