Serotonin type-1D receptor stimulation of A-type K+ channel decreases membrane excitability through the protein kinase A- and B-Raf-dependent p38 MAPK pathways in mouse trigeminal ganglion neurons-Reference-Cited by-同舟云学术

Serotonin type-1D receptor stimulation of A-type K+ channel decreases membrane excitability through the protein kinase A- and B-Raf-dependent p38 MAPK pathways in mouse trigeminal ganglion neurons

Published:2016-08 Issue:8 Volume:28 Page:979-988
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Zhao Xianyang,Zhang Yuan,Qin Wenjuan,Cao Junping,Zhang Yi,Ni Jianqiang,Sun Yangang,Jiang Xinghong,Tao Jin

Funder

National Natural Science Foundation of China

Natural Science Funding for Colleges and Universities in Jiangsu Province

Scientific Research Foundation for the Returned Overseas Chinese Scholars of State Education Ministry

State Key Laboratory of Neuroscience of Chinese Academy of Sciences

333 High Level Talent Cultivation Project of Jiangsu Province

The Second Affiliated Hospital of Soochow University Preponderant Clinic Discipline Group Project Funding

Qing-Lan Project of JiangSu Province

Dong-Wu Scholar Funding of Soochow University

Priority Academic Program Development of Jiangsu Higher Education Institutions

Publisher

Elsevier BV

Subject

Cell Biology

Reference59 articles.

1. Peripheral pain is enhanced by insulin-like growth factor 1 through a G protein–mediated stimulation of T-type calcium channels

2. KCNQ1, KCNE2, and Na + -Coupled Solute Transporters Form Reciprocally Regulating Complexes That Affect Neuronal Excitability

3. Insulin-Like Growth Factor-1 Receptor-Mediated Inhibition of A-type K+ Current Induces Sensory Neuronal Hyperexcitability Through the Phosphatidylinositol 3-Kinase and Extracellular Signal-Regulated Kinase 1/2 Pathways, Independently of Akt

4. Differential contribution of Kv4-containing channels to A-type, voltage-gated potassium currents in somatic and visceral dorsal root ganglion neurons

5. Enhanced Excitability and Down-Regulated Voltage-Gated Potassium Channels in Colonic DRG Neurons from Neonatal Maternal Separation Rats

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