The interruption of atypical PKC signaling and Temozolomide combination therapy against glioblastoma-Reference-Cited by-同舟云学术

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The interruption of atypical PKC signaling and Temozolomide combination therapy against glioblastoma

Published:2021-01 Issue: Volume:77 Page:109819
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Dey Avijit,Islam S.M. Anisul,Patel Rekha,Acevedo-Duncan Mildred

Funder

William and Ella Owens Medical Research Foundation

Musella Foundation

Bradley Zankel Foundation, Inc

Publisher

Elsevier BV

Subject

Cell Biology

Reference42 articles.

1. Glioblastoma multiforme: the terminator;Holland;Proc. Natl. Acad. Sci. U. S. A.,2000

2. Glioblastoma multiforme - an overview;Urbanska;Wspolczesna Onkol,2014

3. The definition of primary and secondary glioblastoma;Ohgaki;Clin. Cancer Res.,2013

4. Definition of primary and secondary glioblastoma - letter;Werner;Clin. Cancer Res.,2014

5. Genetics and epigenetics of glioblastoma: applications and overall incidence of IDH1 mutation;Liu;Front. Oncol.,2016

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1. PTX3 promotes breast cancer cell proliferation and metastasis by regulating PKCζbreast cancer, pentraxin 3, protein kinase Cζ, proliferation, metastasis;Experimental and Therapeutic Medicine;2024-02-01

2. Dual inhibition of atypical PKC signaling and PI3K/Akt signaling dysregulates c-Myc to induce apoptosis in clear cell Renal Cell Carcinoma;Frontiers in Oncology;2024-01-15

3. Non-coding RNAs (ncRNAs) and multidrug resistance in glioblastoma: Therapeutic challenges and opportunities;Pathology - Research and Practice;2024-01

4. Insulin/IGF Axis and the Receptor for Advanced Glycation End Products: Role in Meta-inflammation and Potential in Cancer Therapy;Endocrine Reviews;2023-03-04

5. ANXA5 promotes glioma progression through the FAk/PI3K/AKT pathway;2022-11-30

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