Bone morphogenetic protein-9 activates Smad and ERK pathways and supports human osteoclast function and survival in vitro-Reference-Cited by-同舟云学术

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Bone morphogenetic protein-9 activates Smad and ERK pathways and supports human osteoclast function and survival in vitro

Published:2013-04 Issue:4 Volume:25 Page:717-728
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Fong David,Bisson Martine,Laberge Gino,McManus Stephen,Grenier Guillaume,Faucheux Nathalie,Roux Sophie

Funder

Natural Sciences and Engineering Research Council of Canada (NSERC)

Canada Institute of Health Research (CIHR)

FRSQ (Fonds de la Recherche en Santé du Québec)

Publisher

Elsevier BV

Subject

Cell Biology

Reference56 articles.

1. Bone Morphogenetic Proteins: A critical review

2. TGFβ–SMAD signal transduction: molecular specificity and functional flexibility

3. Homomeric and heteromeric complexes among TGF-β and BMP receptors and their roles in signaling

4. A homozygous BMPR1B mutation causes a new subtype of acromesomelic chondrodysplasia with genital anomalies

5. A recurrent mutation in the BMP type I receptor ACVR1 causes inherited and sporadic fibrodysplasia ossificans progressiva

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4. Bone morphogenetic protein (BMP)9 in cancer development: mechanistic, diagnostic, and therapeutic approaches?;Journal of Drug Targeting;2023-07-19

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