miR-16 targets Bcl-2 in paclitaxel-resistant lung cancer cells and overexpression of miR-16 along with miR-17 causes unprecedented sensitivity by simultaneously modulating autophagy and apoptosis-Reference-Cited by-同舟云学术

miR-16 targets Bcl-2 in paclitaxel-resistant lung cancer cells and overexpression of miR-16 along with miR-17 causes unprecedented sensitivity by simultaneously modulating autophagy and apoptosis

Published:2015-02 Issue:2 Volume:27 Page:189-203
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Chatterjee Abhisek,Chattopadhyay Dhrubajyoti,Chakrabarti Gopal

Funder

Department of Biotechnology, Govt. of India

Publisher

Elsevier BV

Subject

Cell Biology

Reference62 articles.

1. miR-17-5p Downregulation Contributes to Paclitaxel Resistance of Lung Cancer Cells through Altering Beclin1 Expression

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3. miR-135a contributes to paclitaxel resistance in tumor cells both in vitro and in vivo

4. Paclitaxel induced apoptosis in breast cancer cells requires cell cycle transit but not Cdc2 activity

5. Paclitaxel (Taxol)

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