Distinct roles for mitogen-activated protein kinase phosphatase-1 (MKP-1) and ERK-MAPK in PTH1R signaling during osteoblast proliferation and differentiation
Author:
Funder
National Institute of Health
Publisher
Elsevier BV
Subject
Cell Biology
Reference54 articles.
1. Parathyroid hormone is essential for normal fetal bone formation
2. Lethal skeletal dysplasia from targeted disruption of the parathyroid hormone-related peptide gene.
3. Ablation of the PTHrP gene or the PTH/PTHrP receptor gene leads to distinct abnormalities in bone development
4. Targeted overexpression of parathyroid hormone-related peptide in chondrocytes causes chondrodysplasia and delayed endochondral bone formation.
5. Activated parathyroid hormone/parathyroid hormone–related protein receptor in osteoblastic cells differentially affects cortical and trabecular bone
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