Bcl-3, induced by Tax and HTLV-1, inhibits NF-κB activation and promotes autophagy-Reference-Cited by-同舟云学术

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Bcl-3, induced by Tax and HTLV-1, inhibits NF-κB activation and promotes autophagy

Published:2013-12 Issue:12 Volume:25 Page:2797-2804
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Wang Jinheng,Niu Zhiguo,Shi Ying,Gao Cai,Wang Xia,Han Jingxian,Li Junying,Gao Zhitao,Zhu Xiaofei,Song Xiangfeng,Qin Zhihai,Wang Hui

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Cell Biology

Reference47 articles.

1. Human T-cell leukemia virus type I Tax activation of NF-kappa B/Rel involves phosphorylation and degradation of I kappa B alpha and RelA (p65)-mediated induction of the c-rel gene.

2. Mechanisms of Persistent NF-κB Activation by HTLV-I Tax

3. Effect of propolis and caffeic acid phenethyl ester (CAPE) on NFκB activation by HTLV-1 Tax

4. Ubiquitin-Specific Peptidase 20 Targets TRAF6 and Human T Cell Leukemia Virus Type 1 Tax To Negatively Regulate NF- B Signaling

5. Role of Adapter Function in Oncoprotein-mediated Activation of NF-κB

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3. Virus, Exosome, and MicroRNA: New Insights into Autophagy;Advances in Experimental Medicine and Biology;2022

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5. Transcription Coactivator BCL3 Acts as a Potential Regulator of Lipid Metabolism Through the Effects on Inflammation;Journal of Inflammation Research;2021-09

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