In vivo activating transcription factor 3 silencing ameliorates the AMPK compensatory effects for ER stress-mediated β-cell dysfunction during the progression of type-2 diabetes-Reference-Cited by-同舟云学术

In vivo activating transcription factor 3 silencing ameliorates the AMPK compensatory effects for ER stress-mediated β-cell dysfunction during the progression of type-2 diabetes

Published:2013-12 Issue:12 Volume:25 Page:2348-2361
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Kim Ji Yeon,Park Keun Jae,Kim Gyu Hee,Jeong Eun Ae,Lee Dae Yeon,Lee Seong Su,Kim Dae Jin,Roh Gu Seob,Song Jihyun,Ki Sung Hwan,Kim Won-Ho

Funder

Korean National Institutes of Health

Publisher

Elsevier BV

Subject

Cell Biology

Reference45 articles.

1. Metabolic and genetic influence on glucose metabolism in type 2 diabetic subjects—experiences from relatives and twin studies

2. The Unfolded Protein Response: A Pathway That Links Insulin Demand with β-Cell Failure and Diabetes

3. Beta-cell apoptosis in the pathogenesis of human type 2 diabetes mellitus

4. Disordered Lipid Metabolism and the Pathogenesis of Insulin Resistance

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