Critical role of FANCC in JAK2 V617F mutant-induced resistance to DNA cross-linking drugs-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Critical role of FANCC in JAK2 V617F mutant-induced resistance to DNA cross-linking drugs

Published:2013-11 Issue:11 Volume:25 Page:2115-2124
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Ueda Fumihito,Sumi Kazuya,Tago Kenji,Kasahara Tadashi,Funakoshi-Tago Megumi

Funder

MEXT

Takeda Science Foundation

Uehara Memorial Foundation

Keio Gijuku Fukuzawa Memorial Fund for the Advancement of Education and Research

Publisher

Elsevier BV

Subject

Cell Biology

Reference31 articles.

1. Jaks and Stats in signaling by the cytokine receptor superfamily

2. Jak2: normal function and role in hematopoietic disorders

3. JAK/STAT Pathways in Cytokine Signaling and Myeloproliferative Disorders: Approaches for Targeted Therapies

4. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera

5. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis

Cited by 9 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. DNA polymerase theta protects leukemia cells from metabolic-induced DNA damage;Blood;2022-12-29

2. Pharmacological Modulation of the Crosstalk between Aberrant Janus Kinase Signaling and Epigenetic Modifiers of the Histone Deacetylase Family to Treat Cancer;Pharmacological Reviews;2022-12-08

3. Activation of PI3K/AKT Pathway Is a Potential Mechanism of Treatment Resistance in Small Cell Lung Cancer;Clinical Cancer Research;2021-12-17

4. Methotrexate significantly induces apoptosis by inhibiting STAT3 activation in NPM-ALK-positive ALCL cells;Biochemical Pharmacology;2019-12

5. Phosphorylated CIS suppresses the Epo or JAK2 V617F mutant-triggered cell proliferation through binding to EpoR;Cellular Signalling;2017-02

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3