Cellular defects by deletion of ANO10 are due to deregulated local calcium signaling-Reference-Cited by-同舟云学术

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Cellular defects by deletion of ANO10 are due to deregulated local calcium signaling

Published:2017-01 Issue: Volume:30 Page:41-49
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Wanitchakool Podchanart,Ousingsawat Jiraporn,Sirianant Lalida,Cabrita Inês,Faria Diana,Schreiber Rainer,Kunzelmann Karl

Funder

DFG

Cystic Fibrosis Trust

Publisher

Elsevier BV

Subject

Cell Biology

Reference45 articles.

1. TMEM16A confers receptor-activated calcium-dependent chloride conductance;Yang;Nature,2008

2. Expression cloning of TMEM16A as a calcium-activated chloride channel subunit;Schroeder;Cell,2008

3. TMEM16A, a membrane protein associated with calcium-dependent chloride channel activity;Caputo;Science,2008

4. Calcium-dependent phospholipid scrambling by TMEM16F;Suzuki;Nature,2010

5. Anoctamins are a family of Ca2+ activated Cl- channels;Tian;J. Cell Sci.,2012

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1. Understanding coenzyme Q;Physiological Reviews;2024-10-01

2. The ion channel Anoctamin 10/TMEM16K coordinates organ morphogenesis across scales in the urochordate notochord;PLOS Biology;2024-08-22

3. TMEM16 proteins: Ca²⁺‑activated chloride channels and phospholipid scramblases as potential drug targets (Review);International Journal of Molecular Medicine;2024-07-29

4. Anoctamins in epithelial transport;Cell Calcium;2024-06

5. Epithelial Anoctamins;Cell Calcium;2024-06

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