7,8,3′-Trihydroxyflavone prevents doxorubicin-induced cardiotoxicity and mitochondrial dysfunction via activating Akt signaling pathway in H9c2 cells-Reference-Cited by-同舟云学术

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7,8,3′-Trihydroxyflavone prevents doxorubicin-induced cardiotoxicity and mitochondrial dysfunction via activating Akt signaling pathway in H9c2 cells

Published:2023-12 Issue: Volume:112 Page:110924
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Zhao Jing,Yu Hua-qing,Ge Feng-qin,Zhang Man-ru,Song Yu-chen,Guo Dan-dan,Li Qi-hang,Zhu Hua,Hang Peng-zhou

Funder

Wu Jieping Medical Foundation

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Cell Biology

Reference41 articles.

1. Preventing and treating anthracycline cardiotoxicity: new insights;Sawicki;Annu. Rev. Pharmacol. Toxicol.,2021

2. Mitochondrial quality control mechanisms as therapeutic targets in doxorubicin-induced cardiotoxicity;Wu;Trends Pharmacol. Sci.,2022

3. Mitochondrial determinants of doxorubicin-induced cardiomyopathy;Wallace;Circ. Res.,2020

4. Doxorubicin induces cardiomyocyte death owing to the accumulation of dysfunctional mitochondria by inhibiting the autophagy fusion process;Toda;Free Radic. Biol. Med.,2023

5. Mfn2-mediated mitochondrial fusion alleviates doxorubicin-induced cardiotoxicity with enhancing its anticancer activity through metabolic switch;Ding;Redox Biol.,2022

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1. BDNF mimetics recover palmitic acid-induced injury in cardiomyocytes by ameliorating Akt-dependent mitochondrial impairments;Toxicology and Applied Pharmacology;2024-05

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