Protocol for the isolation and purification of endoplasmic reticulum-plasma membrane junctions from the mouse brain
Author:
Funder
NIH
National Tay-Sachs and Allied Diseases Association
Assisi Foundation of Memphis
American Lebanese Syrian Associated Charities
Publisher
Elsevier BV
Reference5 articles.
1. Altered GM1 catabolism affects NMDAR-mediated Ca(2+) signaling at ER-PM junctions and increases synaptic spine formation in a GM1-gangliosidosis model;Weesner;Cell Rep.,2024
2. Glycosphingolipids within membrane contact sites influence their function as signaling hubs in neurodegenerative diseases;Weesner;FEBS Open Bio,2023
3. The isolation of nerve endings from brain: an electron-microscopic study of cell fragments derived by homogenization and centrifugation;Gray;J. Anat.,1962
4. Isolation of Mitochondria-Associated ER Membranes (MAMs), Synaptic MAMs, and Glycosphingolipid Enriched Microdomains (GEMs) from Brain Tissues and Neuronal Cells;Annunziata;Methods Mol. Biol.,2021
5. Generalized CNS disease and massive GM1-ganglioside accumulation in mice defective in lysosomal acid beta-galactosidase;Hahn;Hum. Mol. Genet.,1997
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