Adenosine 3′,5′-cyclic monophosphate-mediated prolactin secretion in GH4C1 cells involves Ca2+ influx through L-type Ca2+ channels-Reference-Cited by-同舟云学术

Adenosine 3′,5′-cyclic monophosphate-mediated prolactin secretion in GH4C1 cells involves Ca2+ influx through L-type Ca2+ channels

Published:1993-03 Issue:3 Volume:14 Page:219-226
ISSN:0143-4160
Container-title:Cell Calcium
language:en
Short-container-title:Cell Calcium

Author:

Inukai T.,Wang X.,Greer S.E.,Greer M.A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference19 articles.

1. Role of cyclic adenosine 3′,5′-monophosphate-dependent protein kinase in hormone-stimulated beta-endorphin secretion in AtT20 cells;Schecterson;Mol. Endocrinol,1991

2. Intracellular Ca2+-dependent protein kinase C activation mimics delayed effects of thyrotropin-releasing hormone on clonal pituitary cell excitability;Dufy;Endrocrinology,1987

3. Ca2+-calmodulin-dependent-phosphorylation and platelet secretion;Nishikawa;Nature,1980

4. Difference in calcium requirements for forskolin-induced release of prolactin from normal pituitary cells and GH4C1 cells in culture;Delbeke;Endocrinology,1984

5. cAMP- and diacylglycerol-mediated pathways elevate cytosolic free calcium concentration via dihydropyridine-sensitive, omega-conotoxin-insensitive calcium channels in normal rat anterior pituitary cells;Schofl;Naunyn Schmiedebergs Arch. Pharmacol.,1989

Cited by 7 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Cilostazol, an Inhibitor of Type 3 Phosphodiesterase, Stimulates Large-Conductance, Calcium-Activated Potassium Channels in Pituitary GH3Cells and Pheochromocytoma PC12 Cells;Endocrinology;2004-03

2. Direct effects of prolactin on corticosterone release by zona fasciculata-reticularis cells from male rats;Journal of Cellular Biochemistry;1999-06-15

3. Cellular Mechanisms of Melatonin Action;Physiological Reviews;1998-01-07

4. Cathepsin B Is a Prorenin Processing Enzyme;Hypertension;1996-03

5. Caffeine-induced oscillations of cytosolic Ca2+ in GH3 pituitary cells are not due to Ca2+ release from intracellular stores but to enhanced Ca2+ influx through voltage-gated Ca2+ channels;Pflügers Archiv European Journal of Physiology;1996-01